Scientific research on nutrition and natural approaches and resources in cancer. Studieabstracten study and analysis.
Here we list articles and studies or research on cancer in relation to nutrition. With some additional comments other as we found on the Internet. We will complement these articles regularly, but that takes time and we want very carefully. We have tried to research and articles in logical and more or less alphabetical order. Often an article in the name of nutrient - vitamin etc. But sometimes in diets or diet in cancer or cancer type out the first named, including bladder cancer and vitamins in the BA. So take the name of one nutrient or use search to start because of various cancers are also often important studies and articles that are not all in this list.
If there are people who can give us tips like please send us remarkable investigations and we post on the site. (See also reading list ( This list compiled by physician-biologist Erik Valstar has exceeded 1500 and are all randomized trials) and cancer rates ). In addition, a recommendation to information on proven effects of diet and nutritional supplementation in cancer of the book: Nutritional intervention in cancer, a signal to the conventional oncology. Because physician-biologist Dr Engelbert Valstar. The book costs 16.95 and the ISBN number is 90 5860 192 7.
DMSO (Dimethyl Sulphoxide) proves its worth in studies of various cancers
Doctor Valstar reported earlier on the page bibliography (numbers 112, 113 and 114) that it is not toxic agent DMSO (dimethyl sulphoxide) to include gastric cancer and indicates significant improvement.
On this website has a thing or two about DMSO to read a short quotation translated by Ed thanks for that (see his story how he Crohn's disease below the knee was), but go to the English website for more and clearer information about DMSO:
The DMSO provides the cancer cells (and surrounding cells) from an alternative source of oxygen.
DMSO thus serves as a backup when the hemoglobin oxygen transport does not work properly.
The hemoglobin does not work well with cancer because cancer cells have an anaerobic metabolism and excrete any CO2 ..
Hemoglobin for the transport of O2 depends on CO2.
Because the defective hemoglobin O2 transport "choke" other nearby cells and changes in cancer cells ..
DMSO, however, provides for the O2 and thus ensures that no other cells turn into cancer cells and also that cancer cells change back into semi-healthy cells.
This semi-healthy cells commit suicide over time, like every healthy cell. This mechanism is turned off in cancer cells ..
Below are four different studies suggest that DMSO also shows improvement in prostate cancer and melanoma. The principle of operation of DMSO based on the same operation that other resources on the 'telomerase' effect on cancer cells, such as angiostatin and endostatin. It is important that DMSO inhibits the growth of cancer cells or stop, allowing the cancer cells die naturally without side effects and toxicity. It is not the conclusion that it would solve. This requires much more research is needed according to the experts. Medline But even these studies and present record does indicate that DMSO is a credible way.
REFERENCE 1
MEDLINE Title: Stimulation of differentiation in human melanoma cells by dimethyl sulphoxide (DMSO).
Author Siracký J, M Blasko, J Borovanský Address Source Neoplasm, 1985, 32:6, 685-8 Abstract
Stimulation of differentiation in human amelanotic melanoma cell line by 0.5, 1, 1.5 and 2% DMSO overexpressed in Increased melanization was significantly linked to Decrease in cell proliferation rate or thesis. After Removing the 0.5 and 1% DMSO samples Became Within 14 Days With The Same Again amelanotic growth capacity as untreated controls. The 1.5 and 2% samples did not recover after Removing DMSO and the inhibition of proliferation was irreversible.
Language of Publication English Unique Identifier 86118891
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Reference 2
the effect of DMSO on Prostate cancer .........
Prostate 1989, 15 (2) :123-33
Effect of DMSO and DFMO on rat prostate tumor growth.
Carvalho L, Foulkes K, Mickey DD
Department of Surgery, University of North Carolina, Chapel Hill
27599-7235.
An anaplastic, meta static subline of the Dunning rat tumor was Exposed to non-cytodestructive doses of the cellular differentiation agents
dimethylsulfoxide and difluoromethylornithine. Results showed significantly Slower solid tumor growth after .... oral treatment of host animals for 20 days before injection with Either Agent or untreated tumor cells, and after oral treatment of host
Either agent initiated with animals on the day of untreated tumor cell injection.
Treatment of animals with tumors with Either Agent Established Also had an inhibitory effect on tumor growth, and the effect was related to the length of treatment. Galanthus, or exposure hypothesis highly malignant rat prostate carcinoma cells to non-cytotoxic doses or Slower Either agent induced tumor growth rates. Since a slowing of cell cycle transit times is an early indicator of cellular differentiation, synthesis results reflect Could an Increase in the capacity of the malignant cells to differentiation (to become non-cancerous cells).
PMID: 2508071, UI: 90017079
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Reference 3
Title
Oxygen-derived free-radical scavengers ProLong survival in gastric cancer.
Author Salim AS
Chemotherapy, 1992, 38:2, 135-44
Abstract
Following Potentially Curative distal two-thirds partial gastrectomy, 228 patients making an uneventful recovery from surgery Were randomized to the control group or to receive allopurinol (50 mg by mouth 4 times a day) or DMSO (500 mg by mouth 4 times a day) . In 160 fully evaluable Patients who Were studied for 5 years, allopurinol and DMSO Incurred a significant (p Less Than 0.01) survival advantage over the whole period of study.
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Reference 4
Title
Dimethyl sulfoxide (DMSO) Causes a reversible inhibition
or telomerase activity in a Burkitt lymphoma cell line.
Author
Sharma S, Raymond E, Soda H; Izbicka E, Davidson K;
Lawrence R, Von Hoff DD
Source
Leuk Res, August 1998, 22:8, 663-70
INTRODUCTION: Telomerase is an enzyme is required for maintenance or That telomeres. This enzyme Has Been Shown to be present in germline tissues and tumors and tumor cell Majority or lines. The regulation of telomerase is an active area or different models becauses Investigation, Potentially, inhibition of this enzyme could-be important in cancer therapy. To study the regulation of this enzyme in lymphoma cell lines, we DMSO Used to produce a Reversible G0/G1 arrest in Raji cell line, as Shown EARLIER [Sawai M, Takase K, Teraoka H, Tsukada K. Reversible G1 arrest in the cell cycle of human lymphoid cell lines by dimethyl sulphoxide. Exp Cell Res 1990, 187:4-10].
RESULTS: In this model, DMSO reversibly inhibited
That Could Be restored telomerase activity after release from the
blockage. The inhibition of telomerase Seems to parallel cellular
proliferation and it Appears That telomerase is regulated upon entry Into the cell cycle.
CONCLUSION:
Our observations Demonstrate a novel effect of DMSO on cellular mechanisms in Raji cell line (Lymphoma Cells).
Unique Identifier 98343686
