Kanker-actueel Sunlight - (vitamin D) protects against many cancers (25% to 40%), including lymklierkanker (non-Hodgkin's) and even non-melanoma skin cancers.

Lymphoma. Hodgkinlymfomen and non-Hodgkin's disease

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Sunlight - (vitamin D) protects against many cancers (25% to 40%), including lymklierkanker (non-Hodgkin's) and even non-melanoma skin cancers.

May 15, 2007: Source: 1: Eur J Cancer. 2007 May 29; Others

Several studies show that sunlight protects against cancer. Even non-Hodgkin's and non-melanoma skin cancers are less common in people who are within a normal pattern of exposure to sunlight. All researchers suggest that sunlight stimulates the body to produce Vitamin D and vitamin D protects against cancer. Even intake of vitamin D in the form of a dietary supplement protects against cancer. We have a number of recent studies, but who put further searches in PubMed can find much more.

Does solar exposure, as Indicated by the non-melanoma skin cancers, protect from solid cancers: Vitamin D as a Possible Explanation. Tuohimaa P, Pukkala E, Scelo G, Olsen JH, Brewster DH, Hemminki K, Tracey E, Weiderpass E, Kliewer EV, Pompe-Kirn V, McBride ML, Martos C, Chia KS, Tonita JM, Jonasson JG, Boffetta P, Brennan P.
Medical School, Department of Clinical Chemistry, University of Tampere, Tampere University Hospital, 33014 Tampere, Finland.

BACKGROUND: Skin cancers are Known to be associated with sun exposure, whereas sunlight through the production of vitamin D May protect against some cancers. The AIM of this study was to Assess whethere patiënten with skin cancer have an altered risk of Developing Other Cancers.

METHODS: The study cohort consisted of 416,134 cases of skin cancer and 3,776,501 cases of non-skin cancer as a first cancer Extracted from 13 cancer registries. 10.886 35.620 melanoma and non-melanoma skin cancer cases had second cancers. The observed numbers (O) or type 46 or a second primary cancer after skin melanoma, basal cell carcinoma or non-basal cell carcinoma, and skin cancers or skin cancers following non-Were compared to the expected numbers (E) derived from the age, sex and calendar period specific cancer incidence rates in Each of the cancer registries (O / E = SIR, standardized incidence ratios). Rates from cancer registries are classified to sunny Countries (Australia, Singapore and Spain) and less sunny Countries (Canada, Denmark, Finland, Iceland, Norway, Scotland, Slovenia and Sweden) Were compared to eachother.

RESULTS: SIR of all second solid primary cancers (except skin and lip) after skin melanoma Were significantly lower for the sunny Countries (SIR (S) = 1.03, 95% CI 0.99-1.08) in the Less Than Sunny Countries (SIR (L ) = 1.14, 95% CI 1.11-1.17). The difference was more obvious after non-melanoma skin cancers: basal cell carcinoma after SIR (S) / SIR (L) = 0.65 (95% CI = 0.58-0.72), basal cell carcinoma after non-SIR (S) / SIR ( L) = 0.58 (95% CI = 0.50-0.67). Countries in sunny, the risk of second primary cancer after non-melanoma skin cancers was lower for cancers of the musts for Forman lip, mouth and non-Hodgkin's lymphoma.

CONCLUSIONS: Vitamin D production in the skin Seems to Decrease the Risk of Several solid cancers (Especially stomach, colorectal, liver and gallbladder, pancreas, lung, female breast, prostate, bladder and kidney cancers). The apparently protective effect of sun exposure against second primary cancer is more pronounced after non-melanoma skin cancers Than Melanoma, Which is consistent with reports EARLIER That non-melanoma skin cancers reflect cumulative sun exposure, whereas melanoma is more related to sunburns.
PMID: 17540555 [PubMed - as supp song by publisher]

1: Cancer Epidemiol Biomarkers Prev. March 2007, 16 (3) :396-400. Epub 2007 Mar 2.

Sun exposure and non-Hodgkin's lymphoma.
Armstrong BK, Kricker A.
Sydney Cancer Centre, Royal Prince Alfred Hospital, Missenden Road, Camperdown 2050, Sydney, New South Wales, Australia. brucea@health.usyd.edu.au

It was initially hypothesized That Might sun exposure cause non-Hodgkin's lymphoma (NHL) on the following backgrounds: its incidence was in parallel with Increasing Cutaneous melanoma or That, ITS was Increased Risk In Those with a history of melanoma skin cancer or Other; sun Exposure Causes immune suppression, and Other Reasons for immunosuppression is associated with an Increased risk of NHL. The association of NHL with prior skin cancer Has Been found consistently in subsequent component studies, but results of ecological assessments have only partially-supported this hypothesis. Contrary to it, three recent studies of NHL found Individuals That Decreased risk, gene rally by 25% to 40%, across categories of Increasing total or recreational, but not occupational, sun exposure. One study, Galanthus far Reported only in abstract, showed the opposite. Production of vitamin D from sun exposure, offering a plausible mechanism for protection against NHL by sun exposure. HAS A recent study found a Reduced Risk of NHL in people with a high dietary intake of vitamin D. Results of Additional Studies in Individual and a planned original-data meta-analysis of case-control studies Should help to resolve the present conflict has resulted on sun exposureand NHL.

PMID: 17337644 [PubMed - indexed for MEDLINE]

1: Int Arch Occup Environ Health. August 2007, 80 (8) :663-70. Epub 2007 Mar 2.

Occupational exposure to ionizing and non-ionizing radiation and risk of non-Hodgkin's lymphoma. Karipidis KK, Benke G, Sim MR, Kauppinen T, Kricker A, Hughes AM, Grulich AE, Vajdic CM, Kaldor J, Armstrong B, Fritschi L. Department of Epidemiology and Preventive Medicine, Monash University, Melbourne, VIC, Australia. OBJECTIVE: To Investigate the Association Between occupational exposure to ionizing, ultraviolet (UV), radio frequency (RF) and Extremely Low Frequency (ELF) Radiation and Risk of Developing Non-Hodgkin's lymphoma (NHL) in a population-based case-control study.

METHODS: The study population consisted of 694 NHL cases, first diagnosed Between 1 January 2000 and 31 August 2001, and 694 controls from two regions in Australia, matched by age, sex and region of residence. A detailed occupation history was first Obtained Using a lifetime calendar and a telephone interview. Exposure to radiation was then assessed Using a Finnish job-exposure matrix (FINJEM). Odds ratios (ORs) and 95% confidence intervals (CIs) Were Calculated from logistic regression models That included the matching variables as Covariates.

RESULTS: For ionizing radiation, the ORs Were close to unity. For UV and ELF radiation, The Highest Exposed group of workers had ORs of 1.32 (95% CI = 0.96-1.81) and 1.25 (95% CI = 0.91-1.72), respectively. For UV radiation there was a positive dose-response When exposure was lagged by 5 and 10 years (P for trend 0.04 for Both lag period). Workers in the upper tertile of exposure for RF radiation had an OR of 3.15 (95% CI = 0.63-15.87), but the estimate was based on very small numbers. CONCLUSIONS: Our results do not Provide Support for an Association Between NHL and occupational exposure to ionizing or ELF radiation. For UV radiation, our Findings are consistent with a weak positive association. Further investigation focusing on UV and RF radiation and NHL is required.

PMID: 17334774 [PubMed - in process]

Epidemiology Sun Exposure May Protect Against Non-Hodgkin's lymphoma: A case-control study
Ann Maree Hughes 1, Bruce K. Armstrong 1, Claire M. Vajdic 2, 3 Jennifer Turner, Andrew E. Grulich 2, 4 Lin Fritschi, Sam Milliken 3, John Kaldor 2, Geza Benke 5, 1 * Anne Kricker 1School of Public Health, The University of Sydney, Australia 2National Centre for HIV Epidemiology and Clinical Research, University of NSW, Sydney, Australia 3pcs. Vincent's Hospital, Sydney, Australia 4School of Population Health, The University of Western Australia, Perth, Australia 5Department of Epidemiology and Preventive Medicine, Monash University, Melbourne, Australia

email: Anne Kricker (annek@health.usyd.edu.au)

* Correspondence to Anne Kricker, School of Public Health, Medical Foundation Building (K25), University of Sydney, NSW 2006, Australia Fax: +612-9036-3168

Funded by:

National Health and Medical Research Council, Australia, Grant Number: 990920

Keywords
Non-Hodgkin's lymphoma • aetiology • sun exposure • UV Radiation • case-control study

Abstract
Ultraviolet radiation is a hypothesised risk factor for non-Hodgkin's lymphoma (NHL), but no epidemiological study HAS Examined Using this association directly Measures of sun exposure in Individuals. Adults aged 20-74 years living in NSW and ACT, Australia, Were the study population. Cases (704 or 829 Invited to take part, 85%) Were diagnosed January 2000 to August 2001. Controls (694 or 1136 Invited to take part, 61%) Were Randomly selected from state Electoral rolls and frequency-matched to cases by age, sex and state of residence. A Self-Administered Questionnaire and telephone interview Measured outdoor hours on working and nonworking days and vacations at 10, 20, 30, 40, 50 and 60 years of age. Logistic regression models of NHL and sun exposure contained the three matching variables, ethnicity and sun sensitivity Measures as Covariates. Contrary to expectations, with risk of NHL Fell Reported Increasing sun exposure hours. Relative to 1.0 for the lowest quarter of total sun exposure hours, the odds ratios (ORs) for successively higher quarters Were 0.72 (95% CI 0.53-0.98), 0.66 (0.48-0.91) and 0.65 (0.46-0.91) (P trend = 0.01). The association of sun exposure on nonworking days with NHL were stronger, OR for Highest quarter 0.47 (0.34-0.66) (P trend = 0.0001). Also Fell risk with sun exposure on vacations; Highest quarterback for OR 0.60 (0.43-0.85) (P trend = 0.003). These associations appeared strongest in women and in childhood. There was little evident trend in risk with exposure on working day. Our results providence strong statistical evidence for an inverse association Between sun exposure and NHL. Increasing Evidence That Vitamin D May Protect Against Cancer makesUV-mediated synthesis of vitamin D a plausible mechanism whereby sun exposure Might Protect Against NHL. © 2004 Wiley-Liss, Inc..

Received: 16 March 2004, Accepted: 21 May 2004